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These cells appear to contribute to the aging process and age-related diseases through the senescence-associated secretory phenotype (SASP), which fosters a pro-inflammatory environment detrimental to tissue function and integrity
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Connors J, Taramangalam B, Cusimano G, Bell MR, Matt SM, Runner K, et al
Furthermore, cAMP-dependent mechanisms acting through PKA, and the exchange protein directly activated by cAMP (EPAC) inhibit ATP-dependent potassium channels and increase the activity of calcium channels
In mice with ICH, it has been demonstrated that Irisin reduces M1 macrophage levels while promoting an increase in M2 macrophages, thus exhibiting an anti-neuroinflammatory effect
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